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Despite all advances in neuroscience in recent decades, age-associated cognitive decline resulting from neurodegenerative processes in the brain remains a challenge for researchers and clinicians due to the highly complex nature of its pathogenesis

It is activated to its oxidized form in the cytoplasm by NAD(P)H:quinone oxidoreductase (NQO1), allowing shuttle of electrons directly to complex III, bypassing the malfunctioning complex I