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With regard to the molecular toxic mechanisms of tobacco, oxidative stress or inflammasome activation as well as a chronic activation of aryl hydrocarbon receptor signaling can contribute to premature aging and the development of neoplasms by affecting metabolism, extracellular matrix remodeling, inflammation, pigmentation, DNA repair, and apoptosis 10

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This led to the following observations: - CYP6B46 doesnt detoxify nicotine Instead, it redirects a small fraction of ingested nicotine from the midgut into the hemolymph - allowing larvae to exhale nicotine through their spiracles