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Treatment with the GIPR antagonist and acyl-GLP-1 both led to a reduction in the likelihood of Nrxn3-Nlgn1 signalling between C35 GABA4 neurons and Pomc.GLU-5 and Agrp.GABA-4 neurons, a change that was specific to these feeding-related neurons and absent in other hypothalamic neurons (Fig

Studies have reported it may speed up the healing of tendons, ligaments, and muscles in research models linked to its ability to increase growth factors involved in connective tissue repair

doi: (Of outstanding importance)

circMAP3K4 regulates insulin resistance in trophoblast cells during gestational diabetes mellitus by modulating the miR-6795-5p/PTPN1 axis