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Furthermore, BTZ treatment in rats caused a decrease in SIRT1 levels, which subsequently reduced STAT3-mediated histone hyperacetylation in the neutrophilic alkaline phosphatase 1 promoter region of dorsal horn neurons, ultimately contributing to the development of mechanical pain hypersensitivity ( 3.6 Central nervous system dysfunction BTZ does not cross the blood-brain barrier, suggesting that it may indirectly contribute to CNS dysfunction (Meregalli et al., 2014)

doi: 10.1038/s41573-019-0036-1 27 BonviciniF.MarcelloN.MandrioliJ.PietriniV.VincetiM

"Expanded Recycling Site Upsets an Ossining Neighborhood"