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RELMs are believed to display inflammation-regulating, chemokine and growth-factor properties [204] and are involved in propagation of oxidative stress [211]

Overexpression of the cell adhesion protein neuroligin-1 induces learning deficits and impairs synaptic plasticity by altering the ratio of excitation to inhibition in the hippocampus

The cancer cachexia model was established by subcutaneously transplanting 5 10 6 CT26 cells into the left flanks of male mice as described in a previous study [31] (as day 0, D0)

No rescuing effects were observed with z-VAD-fmk, suggesting that apoptosis is not induced by GSH depletion