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Nevertheless, downstream analyses identified reproducible differences in specific metabolites that were consistently associated with ASD status within this cohort

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The WD40 domains of COP1 b and COP1 d interact with DET1 in a manner similar to the COP1-DDD-Ube2e dimer (described below), while the RING dimers are in close proximity to Ube2e2 (Fig

Moreover, SIRT1 reduces neuronal injury by inhibiting the expression of inducible nitric oxide synthase (iNOS) which contributes to the aggravation of neuronal injury via the activation of glutamate neurotoxicity (Zhang et al