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Some of these would likely be found at increased frequency in any population of patients with AD, thus potentially constituting biomarkers (perhaps even marking characteristic AD endophenotypes in some cases) [525]

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Central regulators of mitochondrial reprogramming: coordinated control by metabolic enzymes, spatiotemporal heterogeneity, and post-transcriptional networks Building on previous sections that examined how CRC develops resistance through mitochondrial metabolic reprogramming, this section explores three key regulatory mechanisms that sustain such adaptability

Human evidence, regulatory authorisation and online product identity are separate questions