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By inhibiting autophagy, the TP53 mutation, which is less common in hematological cancers than in solid tumors, increases p53 expression and triggers apoptotic responses dependent on p53-upregulated modulator of apoptosis and BAX, leading to AML cell destruction (44)

Recognition of oxidized lipids by macrophages and its role in atherosclerosis development

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Tail spasticity Interestingly, the development of spasticity began earlier in the rats that underwent spinal cord injury and had been treated with BPC 157 than in the corresponding controls