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In another study, Wang et al

A preliminary finding by our team did indicate kidney injury by the two contaminants in combination, which triggers further investigation

The authors noted limitations including reliance on ICD codes to identify NAION, lack of data on diabetes duration prior to Medicare enrollment, and a restricted age range that may limit generalizability

Therefore, early intervention in replenishing mGSH stores by supplying GSH precursor such as N -acetylcysteine (NAC) or targeting upstream mechanisms involved in mitochondrial dysfunction and ROS generation, such as the sustained activation of c-Jun N-terminal kinases (JNK), can result in the protection of APAP-mediated hepatotoxicity