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Minimizing Oxidative Stress in the Lens: Alternative Measures for Elevating Glutathione in the Lens to Protect against Cataract

An exhaustive investigation revealed that an augmentation in ATF4, emanating from the mTORC2-PKC-Nrf2 signaling pathway, culminates in the induction of ATF4 expression and the promotion of Sirt5, a pivotal transcriptional target of ATF4

As GLP1R activation improves oral glucose tolerance via a mechanism independent of insulin and involving inhibition of gastric emptying [23], we assessed gastric emptying via measurement of plasma acetaminophen [24]

This can reduce natural saliva stimulation and alter the pH balance of the mouth