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This dual mechanism results in increased insulin production, decreased glucagon secretion, reduced appetite and energy intake, delayed gastric emptying, and significant weight loss in clinical trials compared to placebo

GI effects Diarrhea (common) Abdominal pain C

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In this Review, we evaluate the translational evidence supporting GLP-1RAs as candidate disease-modifying therapies for NDDs, describe mechanistic pathways linking metabolic dysfunction to neuronal vulnerability, and outline remaining challenges and research priorities in this rapidly evolving field