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4.5.7.8.11.12 Though many potential mechanisms have been proposed by which ALA may improve DPN symptoms, most experimental evidence suggests improvements as a result of reduced oxidative stress, improved nerve blood flow, increased nerve Na+/K+ ATPase activity, and elevated nerve conduction velocity (NVC), which is further supported by a series of human studies and meta-analyses

This work was supported by the Belgian National Funds of Research (FRS-FNRS) (convention J.0009.22)

The link of CVS to cation and mitochondrial function might lead one to propose two different pathways leading to the disorder, if not for the fact that these two concepts are intricately interrelated

Thymosin Alpha-1 is currently not approved by the FDA and it is still being studied in ongoing clinical trials