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Journal of Water Process Engineering, 58 , 104850

Excess ROS then activates the NLRP3 inflammasome in microglia, promoting maturation and release of IL-1 ( 4.3 GSH metabolism: a central hub in mid-late SAE The hippocampus, with its high metabolic demands and low antioxidant capacity, is uniquely vulnerable to septic injury ( A Stat1-Gpx3 hub was identified, where TNF-/IL-1-induced Stat1 activation suppresses Nrf2 (confirmed by reduced hippocampal Nrf2)

Hdac inhibition prevents nf-kappa B activation by suppressing proteasome activity: down-regulation of proteasome subunit expression stabilizes I kappa B alpha

Based on in silico analysis, GPOXs were predicted in chloroplast, mitochondria, cytosol, and ER localizations to synthesise H 2 O 2, and ultimately, this hydrogen peroxide gets converted into H 2 O and O 2 by the catalytic action of catalase and peroxidase